Volume 2

Practical manual of diseases of women and uterine therapeutics : for students and practitioners / by H. Macnaughton-Jones.

  • Macnaughton-Jones, H. (Henry), 1844-1918.
Date:
1904
Catalogue details

Licence: In copyright

Credit: Practical manual of diseases of women and uterine therapeutics : for students and practitioners / by H. Macnaughton-Jones. Source: Wellcome Collection.

    58/522 (page 650)
    (b) Myxomatous connective tissue constitutes the inner surface of the tubal wall, and here also we find an interlacing longitudinal and circular muscle layer. The folds in the muscosa are produced by contractions and elongations of these latter muscle layers, serving to occlude the tube during life. (c) A plexus of blood-vessels, arterial and venous, is found outside the longitudinal layer of the wall. (d) There are also two narrow layers of oblique smooth muscle fibres, traceable from the uterine ostium to the fimbriated extremity, corresponding to the two oblique layers of the uterine parietes. These layers regulate the blood-current in the subjacent arteries and veins. Bland-Sutton thus refers to the changes produced by salpingitis :— ‘ When a healthy Fallopian tube is examined in transverse section by means of a microscope, we distinguish easily the serous and muscular coats of the tube, and, standing upon these, the so-called plicae formed by the mucous coat. Submucous layer. , Ciliated epithelium. Circular muscular fibres. . Longitudinal fibres. Fm. 448.—Normal Fallopian Tube in Section, (x 10 diameters.) (Macalister.) Each plica consists of a delicate framework of connective tissue, fringed with columnar ciliated epithelium on the free surface. Delicate strands of unstated muscle-cells may be distinguished near the base of the epithelium, and m t le middle of the fold are many capillaries. As a rule, a few leucocytes may be seen scattered among the meshes of the connective tissue. When sections are ore pared from tubes which have been for some months past the seat ot salpingitis, the appearances are very different. The plicae are swollen to tv ice or thrice the usual size, and all the details of their structure obscured by an innumerable host of cells of various sizes. In many places the limiting epithelium is lost, in others it can be detected, disturbed and disarrange!, here and there seemingly held in position by some glutinous material. In mild cases this peculiar cell-infiltration is limited, and does not involve the whole plica, but in very diseased specimens the cells are not limited to ie plicie, but involve the muscular coat, and extend into the connective tissue of the mesosalpinx.’